In spite of efforts intended to increase the use of Selective Serotonin Reuptake Inhibitors or SSRI in patients having panic disorder just a reticent boost in their application was seen. Treatment guidelines in using psychotropic drugs for panic disorder have appeared to remain constant over the past decade, with the most commonly used medication being benzodiazepine. Patients utilizing SSRIs do not gain a more positive medical effect than those who use benzodiazepine (Bruce, Vasile, Goisman, Salzman, Spencer, Machan & Keller, 2003).

Very few genetic studies have been conducted for the genetic basis of panic disorders, existing data supports the proposition that these disorders have a distinct genetic factor, some studies have found that the first-degree relatives of individuals with panic disorders are four to eight times more at risk for panic disorders than first degree relatives of other psychiatric patients, however no data indicates an association between a specific chromosomal location or mode of transmission and this particular disorder (Sadock, et al. , 2007).

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Genes principally explain the notable familial aggregation of panic disorders. The part of non shared environmental experience considerably underscores the significance of recognizing reputed environmental risk issues that incline people to anxiety or panic (Hetterna, Neale & Kendler, 2001). Not much advancement in psychotherapy treatments for panic disorder has emerged lately, other than the incorporation of technological equipment into customary psychotherapies and the development of a biweekly 24-session, Panic-Focused Psychodynamic Psychotherapy (PFPP).

Moreover, the occurrence of medical comorbidity makes the recognition, presentation, and management of panic disorder more difficult. Comorbid mood disorders frequently happen and result to an inferior quality of living and heightened impairment (Simon & Fischmann, 2005). Panic disorder treatments need to be given more attention since panic attacks are related with the heightened possibility for young individuals to acquire various mental disorders along the diagnostic spectrum and appears to be linked with substance abuse and anxiety disorders (Goodwin, Lieb, Hoefler, Pfister, Bittner, Beesdo & Wittchen, 2004).

Examination of major external, individual, and familial factors connected to the commencement of panic attacks, is an important path for further research. Resources Aschenbrand, S. G. , Kendall, P. C. , Webb,A. , Safford, S. M. , Flannery-Schroeder, E. , (2003) Is childhood separation anxiety disorder a predictor of adult panic disorder and agoraphobia? A seven-year longitudinal study. Journal of the American Academy of Child and Adolescent Psychiatry. 42 (12):1478-85 Barlow, D. H. , Gorman, J. M. , Shear, M. K. , Woods, S. W., (2000)

Cognitive-behavioral therapy, imipramine, or their combination for panic disorder: A randomized controlled trial. Journal of the American Medical Association. 283 (19):2529-36 Bruce, S. E. , Vasile, R. G. , Goisman, R. M. , Salzman, C. , Spencer, M. , Machan, J. T. , Keller, M. B. ,(2003) Are benzodiazepines still the medication of choice for patients with panic disorder with or without agoraphobia? American Journal of Psychiatry. 160 (8):1432-8 Busch, F. N. , Milrod, B. L. ,& Singer, M. B. , (1999) Theory and technique in psychodynamic treatment of panic disorder. Journal of Psychotherapy Practice and Research. 8:234-242.